GluControl
TM
Formula for Blood Sugar Stabilization
One of the most predictable and universal biochemical changes
that occurs as we grow older is a progressive loss of glucose tolerance,
characterized by prolonged post-meal elevations of glucose and insulin. As
glucose tolerance continues to worsen, it increases our risk for obesity,
hypertension, coronary artery disease and diabetes. Glucontrol is a nutrient
combination designed to improve glucose tolerance, inhibit the evolution of
hyperglycemia (high blood sugar) and prevent the pathogenic changes to
molecules and metabolic pathways that result from hyperglycemia and a
life-long exposure to even normal blood sugar levels. The constituents in
GluControl improve blood sugar regulation and prevent hyperglycemia by:
- improving hepatic and peripheral insulin sensitivity;
- enhancing insulin-mediated uptake of glucose into cells
and glycogen synthesis;
- increasing glucose oxidation and glycogen synthesis and
- increasing insulin sensitivity by enhancing cell receptor
binding of insulin.
Vanadyl Sulfate
- A biologically active form of vanadium that restores
sensitivity of glucose and insulin receptors.
- Can improve glucose tolerance and decrease cholesterol
levels in Type 2 patients and decrease insulin requirements.
Galega officinalis (20%
guanylhydrazine)
- Botanical source for the life extension drug metformin
(GlucoPhage)
- Can decrease the formation of advanced glycosylation end
products
Bitter Melon extract (Momordica
charantia)
- Bitter melon is a tropical fruit that can lower blood
glucose without increasing nsulin.
- In one study, bitter mellon significantly improved glucose
tolerance in diabetics by almost 54%.
Quercetin
- Quercetin is a flavonoid that strongly inhibits aldose
reductase, an enzyme which converts blood glucose into sorbitol, which
is strongly implicated in the complications of diabetes.
- Quercetin also enhances insulin secretion and protects the
beta cells of the pancreas against damage by free radicals.
Pyridoxine
- Pyridoxine (Vitamin B-6) protects against the development
of diabetic neuropathy and other complications of diabetes by inhibiting
the glycosylation of proteins.
N-acetyl cysteine
- N-acetyl cysteine (NAC) is a derivative of the sulfhydryl
amino acid cysteine.
- Cysteine is converted into the important antioxidant,
glutathione. Cysteine, in fact, is considered the rate-limiting factor
in the production of glutathione. NAC has both cytoprotective and
antioxidant effects and prevents complications of diabetes.
Vitamin E
- Vitamin E is the major fat-soluble antioxidant that
improves insulin action and helps prevent long-term complications of
diabetes, especially ardiovascular disease.
- Studies reveal a strong independent association between low
vitamin E status before follow-up and an excess risk of diabetes at four
years.
- For Type 2 diabetics vitamin E supplements reduce oxidative
stress, improve membrane physical characteristics and improve glucose
sensitivity and transport.
Vitamin C (ascorbic acid &
ascorbyl palmitate)
- Vitamin C inhibits sorbitol accumulation and glycosylation
of proteins that cause any complications of diabetes.
- Studies have proven that vitamin C supplements at a dosage
as low as 100 milligrams reduce the accumulation of sorbitol within
cells of Type 1 diabetics.
- Chronic vitamin C administration has shown beneficial
effects upon glucose nd lipid metabolism in patients with NIDDM.
- Combining both fat soluble and water soluble forms of
Vitamin C helps rovide more complete protection against molecular
damage.
Conclusion
The ingredients in GluControlTM improve glucose tolerance,
inhibit the evolution of hyperglycemia and a life-long exposure to even
"normal" blood sugar levels. These ingredients in GluControl
improve blood sugar regulation and prevent hyperglycemia by
- improving hepatic and peripheral insulin sensitivity;
- enhancing insulin-mediated uptake of glucose into cells and
glycogen synthesis;
- increasing glucose oxidation and glycogen synthesis; and
- increasing insulin sensitivity by enhancing cell receptor
binding of insulin.
These actions combine to reduce plasma glucose levels without
abnormal and excessive insulin secretion, while simultaneously decreasing
insulin requirements. These actions also inhibit the development of diabetes
and its complications, including cardiopathy, angiopathy, neuropathy,
retinopathy, nephropathy and other disorders of hyperglycemia.