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Alternative Names Return to top
Intracranial bleedDefinition Return to top
Deep intracerebral hemorrhage is a type of stroke caused by bleeding within the deep structures of the brain (thalamus, basal ganglia, pons, and cerebellum).
Causes Return to top
Deep intracerebral hemorrhage can affect any person regardless of age, sex, or race, though it is more common in older people. It may be caused by head injury (trauma), bleeding into a tumor, problems with blood vessels (such as a cerebral aneurysm or angioma) or blood clotting problems. In some cases, no cause can be found.
Bleeding in the brain irritates the brain tissues, causing swelling (cerebral edema). The blood may collect into a mass (hematoma). Both cerebral edema and the presence of a hematoma within the brain put increasing pressure on the brain tissues and eventually destroy them.
Risk factors for deep intracerebral hemorrhage include:
See also:
Symptoms Return to top
The symptoms vary depending on the location of the bleed and the amount of brain tissue affected. Symptoms most commonly develop suddenly, without warning, often during activity. They may be episodic (occurring and then stopping) or slowly get worse over time.
Exams and Tests Return to top
A neurological exam may suggest increased intracranial pressure or decreased brain functions. The person's specific symptoms can help tell which part of the brain is affected. For example, sudden nausea, vomiting, loss of balance, headache, and a rapid decrease in consciousness can mean a bleed in the cerebellum or brainstem.
An eye exam may show swelling of the optic nerve from pressure in the brain, or there may be changes in eye movement. Abnormal reflexes may be present.
Tests may include:
Treatment Return to top
Deep intracerebral hemorrhage is a severe condition that requires prompt medical attention. It can develop quickly into a life-threatening situation.
Treatment depends on the location, cause, and extent of the hemorrhage.
Surgery may be needed, especially if there is bleeding in the cerebellum. Surgery may also be done to repair or remove structures causing the bleed (such as a cerebral aneurysm or arteriovenous malformation).
Medicines used may include painkillers, corticosteroids or diuretics to reduce swelling, and anticonvulsants to control seizures. Other treatments may be recommended, depending on the condition of the person and the symptoms that develop.
Outlook (Prognosis) Return to top
How well a patient does depends on the size of the hematoma and the amount of swelling.
Recovery may occur completely, or there may be some permanent loss of brain function. Death is possible, and may quickly occur despite prompt medical treatment.
Medications, surgery, or other treatments may have severe side effects.
Possible Complications Return to top
When to Contact a Medical Professional Return to top
Call your health care provider if severe headache with nausea, vomiting, decreased vision, numbness, or tingling occurs.
Go to the emergency room or call 911 if other symptoms of deep intracerebral hemorrhage develop. Emergency symptoms include difficulty breathing, seizures, loss of ability to move or swallow, sudden loss of sensations, sudden change in mental state, and loss of consciousness.
Prevention Return to top
Treatment or control of underlying disorders may reduce the risk of developing intracerebral hemorrhage. High blood pressure should be treated. Do not stop taking medications unless told to do so by your doctor.
Always use safety equipment and take safety precautions in sports, recreation, and work. For example, use hard hats, bicycle or motorcycle helmets, and seat belts. Never dive into water if you do not know the depth of the water or if there may be rocks under the surface.
If you are taking a blood thinner (such as Coumadin), follow your doctor's instructions on how to take the medicine and when to have blood tests for monitoring the medication.
Update Date: 7/14/2006 Updated by: J.A. Lee, M.D., Division of Surgery, UCSF, San Francisco, CA. Review provided by VeriMed Healthcare Network.
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Page last updated: 02 January 2008 |